Program in Gene Function and Expression; Program in Molecular Medicine
Response or Comment
Animals; Diabetes Mellitus, Type 1; Endoplasmic Reticulum; Female; Insulin-Secreting Cells; Stress, Physiological
Endocrine System Diseases | Genetics and Genomics | Immunology and Infectious Disease
Comment on: Tersey SA, Nishiki Y, Templin AT, Cabrera SM, Stull ND, Colvin SC, Evans-Molina C, Rickus JL, Maier B, Mirmira RG. Islet β-cell endoplasmic reticulum stress precedes the onset of type 1 diabetes in the nonobese diabetic mouse model. Diabetes. 2012 Apr;61(4):818-27.
Rights and Permissions
© 2012 by the American Diabetes Association.
Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.
DOI of Published Version
Diabetes. 2012 Apr;61(4):780-1. Link to article on publisher's site
O'Sullivan-Murphy BM, Urano F. (2012). ER stress as a trigger for beta-cell dysfunction and autoimmunity in type 1 diabetes. Program in Gene Function and Expression Publications. https://doi.org/10.2337/db12-0091. Retrieved from https://escholarship.umassmed.edu/pgfe_pp/199