This collection showcases the books, journal articles, book reviews, and other publications and presentations written by faculty and researchers of the Department of Cancer Biology.

Submissions from 2008

New wirings in the survivin networks, Dario C. Altieri

Integrins in prostate cancer progression, Hira Lal Goel, Jing Li, Sophia Kogan, and Lucia R. Languino

Submissions from 2007

Hsp60 regulation of tumor cell apoptosis, Jagadish C. Ghosh, Takehiko Dohi, Byoung Kang, and Dario C. Altieri

FANCJ (BACH1) helicase forms DNA damage inducible foci with replication protein A and interacts physically and functionally with the single-stranded DNA-binding protein, Rigu Gupta, Sudha Sharma, Joshua A. Sommers, Mark K. Kenny, Sharon B. Cantor, and Robert M. Brosh

The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cells, Min Peng, Rachel Litman, Jenny X. Xie, Sudha Sharma, Robert M. Brosh, and Sharon B. Cantor

Submissions from 2006

Multifactorial contributions to an acute DNA damage response by BRCA1/BARD1-containing complexes, Roger A. Greenberg, Bijan Sobhian, Shailja Pathania, Sharon B. Cantor, Yoshihiro Nakatani, and David M. Livingston

Inhibition of BACH1 (FANCJ) helicase by backbone discontinuity is overcome by increased motor ATPase or length of loading strand, Rigu Gupta, Sudha Sharma, Kevin M. Doherty, Joshua A. Sommers, Sharon B. Cantor, and Robert M. Brosh

BACH1 is a DNA repair protein supporting BRCA1 damage response, Min Peng, Rachel Litman, Zhe Jin, G. Fong, and Sharon B. Cantor

Submissions from 2005

Analysis of the DNA substrate specificity of the human BACH1 helicase associated with breast cancer, Rigu Gupta, Sudha Sharma, Joshua A. Sommers, Zhe Jin, Sharon B. Cantor, and Robert M. Brosh

BACH1 is critical for homologous recombination and appears to be the Fanconi anemia gene product FANCJ, Rachel Litman, Min Peng, Zhe Jin, Fan Zhang, Junran Zhang, Simon N. Powell, Paul R. Andreassen, and Sharon B. Cantor

Submissions from 2004

The BRCA1-associated protein BACH1 is a DNA helicase targeted by clinically relevant inactivating mutations, Sharon B. Cantor, Ronny Drapkin, Fan Zhang, Yafang Lin, Juliana Han, Sushmita Pamidi, and David M. Livingston

Submissions from 2001

BACH1, a novel helicase-like protein, interacts directly with BRCA1 and contributes to its DNA repair function, Sharon B. Cantor, Daphne W. Bell, Shridar Ganesan, Elizabeth M. Kass, Ronny Drapkin, Steven R. Grossman, Doke C. R. Wahrer, Dennis C. Sgroi, William S. Lane, Daniel A. Haber, and David M. Livingston

Submissions from 1999

BRCA1, BRCA2, and Rad51 operate in a common DNA damage response pathway, Junjie Chen, Daniel P. Silver, Sharon B. Cantor, David M. Livingston, and Ralph Scully

Submissions from 1998

Stable interaction between the products of the BRCA1 and BRCA2 tumor suppressor genes in mitotic and meiotic cells, Junjie Chen, Daniel P. Silver, Deepika Walpita, Sharon B. Cantor, Adi F. Gazdar, Gail Tomlinson, Fergus J. Couch, Barbara L. Weber, Terry Ashley, David M. Livingston, and Ralph Scully

Submissions from 1996

Evidence for a Ras/Ral signaling cascade, Larry A. Feig, Takeshi Urano, and Sharon B. Cantor

Submissions from 1995

Identification and characterization of Ral-binding protein 1, a potential downstream target of Ral GTPases, Sharon B. Cantor, Takeshi Urano, and Larry A. Feig